Researchers have confirmed that UVB radiation increases love desire in humans. According to an Israeli study, the endocrine system responds to sunlight, including the release of sex hormones. However, the mechanism is not entirely clear.
The Sun Affects the Endocrine System
An Israeli research team led by Carmit Levy of Tel Aviv University has traced the mechanism by which ultraviolet radiation increases love desire in both men and women. The researchers conducted an experiment in which they exposed a total of 32 men and women to UVB, which is part of sunlight, under controlled conditions.
The results were clear. The ultraviolet radiation used increases the love desire of both genders. In addition, aggression also increased in men. All indications are that sunlight, which passes through the skin, interferes with the functioning of human sex hormones.
Key Protein
The ultraviolet component of sunlight acts to increase testosterone levels in men. The sun is also involved in controlling human sexuality. However, the specific mechanisms have not been known so far. Israeli researchers have found that the key, in this case, is the p53 protein. This protein is an important regulator that affects the activity of many other genes.
It participates in cell growth as well as its aging and destruction by cell death. He is also in charge of DNA repairs, including damage from ultraviolet radiation. It is also part of our defense against cancer. Its damage is a common cause of cancer.
It has now been shown that the p53 protein is an important mediator between ultraviolet radiation and human passion. When the researchers turned off the p53 protein in the experimental animals, ultraviolet radiation stopped affecting their sexual appetite.
According to the study, 25 minutes of sunlight is enough but “the skin contains various mechanisms for dealing with radiation from sunlight, and one of these is the p53 protein. We must remember that exposure to UV is dangerous, and can damage the DNA, as in the case of skin cancer,” said professor Levy.
Source: https://www.cell.com/cell-reports/fulltext/S2211-1247(21)01013-5
